Diabetes, a poorly structured diagnosis

Diabetes, a poorly structured diagnosis

Why do so many victims suffer and suffer and die?

JOSÉ DE JESÚS TEJADA MAURY (*)

«If we do not expel the dogma of the academy, we will never defeat obscurantism»

(José Tejada Maury)

According to the infographs of the World Health Organization, there are currently 422 million adults with diabetes in the world, which corresponds to 1 in 11 people.

What is diabetes for the academic world and for society in general?

Diabetes is another concept that needs to be re-evaluated. Academic dogma defines diabetes as an irreversible chronic condition when the body loses its ability to produce enough insulin or to use it effectively.

A reference scale is established according to studies between 70 and 100 milligrams per deciliter; it is considered normal, a level between 100 and 125 milligrams per deciliter, depending on the time of taking food and the time when the tests are done, and a level of 126 milligrams per deciliter to diagnose that diabetes is suffered.

According to the protocol classification there are the following types of diabetes:

Type 1 diabetes: the body does not produce enough insulin.

Type 2 diabetes: the body produces insulin but does not use it properly.

Gestational diabetes: a temporary condition during pregnancy.

We must be careful in these interpretations and consider that if the pancreas supplies the insulin hormone it is in order to catalyze glycolysis, that is; insulin does not regulate the amount of glucose in the bloodstream but accelerates its combustion.

How is the patient determined to classify according to the clinical analysis?

Any health professional can diagnose diabetes, prediabetes, and gestational diabetes through blood tests. Blood tests show if the blood glucose level, also called blood sugar, is too high.

Who should be tested for diabetes?

Anyone who has symptoms of diabetes should be examined to detect the disease. Some people will not have symptoms, but they may have risk factors for diabetes and should be examined. The tests allow health professionals to detect diabetes earlier and work with their patients to manage the disease and prevent its complications.

The tests also allow health professionals to detect prediabetes. Lifestyle changes aimed at losing a moderate amount of weight, if you are overweight, can help to slow or prevent type 2 diabetes.

Diabetes type 1

Testing for type 1 diabetes is almost always done for people who have symptoms of the disease. Doctors often diagnose type 1 diabetes in children and young adults, because type 1 diabetes can be hereditary. A study called Trial Net offers tests to family members of people who suffer from the disease, even if they have no symptoms.

Type 2 diabetes

Experts recommend routine tests to detect type 2 diabetes if you are over 45 years of age, or between 19 and 44 years old and overweight or obese. One or more of the risk factors for diabetes is a woman who has had gestational diabetes.

Although type 2 diabetes occurs more frequently in adults, it can also occur in children. Experts recommend screening children between 10 and 18 who are overweight or obese and at least two of these other risk factors for diabetes: low birth weight; her mother had diabetes during her pregnancy; any risk factor mentioned in the risk factors for type 2 diabetes.

Gestational diabetes

All pregnant women who are not previously diagnosed with diabetes should be screened for gestational diabetes. If you are pregnant, you should have a glucose tolerance test between 24 and 28 weeks of pregnancy.

Do the tests measure blood glucose or the amount of insulin in the blood?

We leave an inconsistency classification type 1 and type 2: according to the first, the body does not produce enough insulin; for the other case, the body produces it but does not know how to use it, that is, there is a scientific vacuum here, because there is no precision in this regard, semantically it seems to redound in the same, that is, a diabetic pleonasm, if it is that this expression can be used for this protocol case.

What is the basis of the academic and scientific world to say that the body does not know how to use insulin? Will there be a laboratory analysis? bioclinical ratorio that has measured the amount of insulin in the blood to be able to affirm that for type 1 diabetes the body does not produce enough insulin? And something more serious: how is it confirmed and explained with elements of judgment to justify that the body does not use insulin properly for type 2 diabetes? The issue is complicated because only the amount of glucose in the blood is being measured and not amount of insulin in blood; therefore, these protocol classifications of these types are inconsistent because we are talking about the study of blood glucose and not insulinometry.

Therefore, everything that has been said and done about diabetes, both the protocol procedures to diagnose and classify the types of diabetes lack scientific basis.

 What would be the way to get oriented in the true cause of the problem?

 We would have to ask ourselves why the amount of glucose in the blood exceeds the standard values ​​and we would have to look for them not in the supply of insulin from the pancreas, but in the mechanism that regulates the transit of glucose from the blood to the place where the glucose receptors are: why is its flow slowed down? Why is blood glucose clogging? Why is your delay, knowing according to the metabolic path that your destination is the liver?

 Glucose is the fuel used by the body, that is, the body's gasoline for cells to do their work: just like a vehicle, it uses fuel for its operation. Following with the metaphor: let's think about the vehicle's carburetor, which is the device in which air and fuel mixtures are made for the combustion of the engine. If the carburetor is saturated with fuel, it drowns (it is a vulgar term used in automobile mechanics), the engine does not start and then the mechanic proceeds to correct the fault allowing the carburetor of the vehicle does not get excess fuel.

 It is similar to the one that occurs in the human body: if the cell gets too much fuel it drowns, it does not work well, this is proven when the patient adopts a change of lifestyles and in their food. Therefore the problem lies in finding the origin of diabetes not in the pancreas, but in the metabolic route of glucose in the blood and its transit between the liver and the kidneys, that is to find out why the abnormal percentage of glucose is maintained (fuel) in the blood, why its circulation is being delayed, what mechanism causes its return to be slow towards the liver. Therefore, we must keep in mind that the liver is the one that removes the excesses of glucose in the blood, this according to the cycle of glucose and the functions of the liver.

 Glucose homeostasis results from the interrelations of metabolic pathways. That is, in the case of diabetes, the mechanisms of glucose entry and exit must be checked and this is done in the liver and not in the pancreas.

It is detected that insulin or oral hypoglycaemic agents ensure that glycolysis is carried out only at cellular level accelerating its combustion, never do so to regulate the amount of glucose in the blood, because excess glucose in the blood is removed by the liver, which is where the glucose receptors are located and thus maintaining homeostasis inside of the glucose cycle adjusting it to the organism's biological clock.

The solution is to allow the return to be done normally not including retardants in our diet as market strategies, since the use and commercialization of artificial sweeteners was authorized in the year 1980 the percentage of diabetics increased by more than 300% in the decade between 1980 and 1990, especially a large market opened to «insulin sint tica produced with biotechnology 'This was established palliative big business for the pharmaceutical industry as a profitable synergy formed. Today the percentage of diabetics, according to the WHO, has increased by 400% globally.

When a patient through alternative therapy is given a supplement, or some therapeutic phyto, it is noted the lowering of blood glucose in the bloodstream , which indicates that the issue is not in the insulin, but we are dealing with a problem at the liver level and that is what we have to analyze to be able to locate the problem well: regulate the glucose output from the bloodstream and not block the pathways metabolic access at the liver level so that its transit through the bloodstream is adjusted to glucose homeostasis and return to the liver without any setback or obstacle. The blood glucose supply of the diabetic patient comes from their food intake due to anxiety (polyphagia), since the high viscosity of blood plasma does not allow nutrients to reach the cell in a normal way. Thirst (polydipsia), is also due to the fact that the cells are dehydrated, due to the same viscosity of the plasma and a lot of urine (polyuria), because the kidney has to excrete the glucose that the liver does not absorb, due to its transit slow in the bloodstream.

How could we solve the problem?

 So that the therapist can guide his analysis in the patient's healing procedure, first of all the patient should not be considered as diabetic, since they have suggested it with "I am diabetic », a phrase that must be modified by« person with slow glucose return », this to be able to locate the origin of the problem well, so that he understands that his problem is not pancreas, but that his organism presents a slowdown within the circulation of the glucose from the blood to the liver (slow return, which is what causes the increase in viscosity). If the problem were insulin, the doses applied would solve the problem of viscosity Blood plasma and the symptoms of this are still experienced by the patient. So, what do they inject the patient with insulin? Could it be that the miscibility of the insulin supplied by the pancreas to the blood is inversely proportional to the viscosity and, as the viscosity increases, the insulin does not reach the cells to fulfill its function as a catalyst? Why is insulin injected into adipose and not intramuscular tissue or into the blood if it is true that insulin lowers the amount of glucose in the blood? It is important to keep in mind that most metabolic disorders are caused by blockages in blood. The intrahepatic ducts, preventing the exit of fluids for digestion, or delaying the excess of glucose in the blood (light diets, the use of medicines, alcohol, preservatives, sweeteners, flavorings). Therefore, hepatic and gall bladder detoxification and cleansing should be guided by the therapist trained to do so, in order to clear the intrahepatic ducts to allow free flow and circulation of the fluids that participate in the processes of metabolism.

Managed with oral hypoglycemic agents and with insulin hormone supply, to get lower values ​​in the bloodstream as a great scientific achievement only from the mathematical (statistical) point of view, never from the epistemological (scientific) point of view.

The initial problem with which the problem of hyperglycemia must have been resolved is to focus on the glucose metabolic pathway, in order to find the root of the problem and not to maintain the disinformation linked to the fear experienced by not having defined a precise definition with analysis. and studies out of all objectivity, intimidating their victims with the diversity of symptoms experienced by those suffering from this disorder derived from the high viscosity in the blood flow, either at the circulatory level, at the vascular level, especially the microcirculation, when diabetic glaucoma occurs, as the aqueous humor can not circulate through the channel Schelemm, which is a small circumferential canal located in the iridocorneal angle of the anterior chamber of the eye, through which drains the aqueous humor into the bloodstream, with which we can affirm that diabetic retinopathy and diabetic glaucoma are not genetic problems. Erectile dysfunction is another one of the symptoms derived from the high viscosity in the blood, the cerebrovascular accidents also obey the high blood viscosity: lack of mobility or resistance to flow and therefore it is not easy to bring nutrients and oxygen to the brain. Or gangrene: having high blood viscosity (resistance to flow), oxygen will not reach the tissues and therefore necrosarán: there is the origin of the terrible diabetic gangrene. We ask: what would be the mechanisms to avoid all these complications and lead a healthy life, without relying on oral or injected hypoglycemic agents?

 The answer: first of all it is necessary to analyze whether the administration of hypoglycaemic drugs suppressed the normal production of insulin by the pancreas, since these chemical molecules are intended to replace a normal function of the hormone-making organ, to supply it artificially, in the bloodstream, limiting the neurotransmitter and synaptic activity between the Peripheral Nervous System and the Central Nervous System, thus blocking the production of part of the pancreas insulin (similar to how to remove the water supply from the aqueduct pipe and sold in containers). Recall once again that the issue is to maintain the normal percentage to avoid the high viscosity in the blood (normal viscosity so that the blood does not show resistance to flow through veins, vessels, capillaries) and fulfills its function of nourishing and oxygenate to the cells of the organism.

For the study a tea was designedtechnique called the Glucose Metabolism Clock, so that each patient manages this mechanism to control its slow return of glucose. As mentioned previously, the accumulation in blood is due to its slow return to the liver, which is the body responsible for removing excess blood glucose. I reiterate my consideration that insulin only helps the combustion of glucose in the cell, this is detected when a patient is given phytotherapeutics (plants, herbs, etc.), without these containing insulin, the low blood glucose in the blood flow is experienced, including patients who injecting insulin, their indicators do not fall below 240, a situation contrary to when they are given by phytotherapeutics, it has been possible to check that it goes down to 90.85.Continuing with the Blood Glucose Metabolism Clock, each patient must consider what kind of food is being supplied to their organism, take into account the glycemic index of their food, that is, how fast a food becomes glucose and how long it passes into the bloodstream. In this, you have to be strict, because according to how you manage your diet, you will manage the presence of blood glucose. To begin with, the patient must have a punctual feeding schedule, that is, he can have breakfast, lunch and dinner with a difference of six hours. For example if you have breakfast at 06:00, have lunch at 12:00 and dinner at 18:00 With the meter you will be taking control the first hour after breakfast, followed by the second hour, until the hour of the lunch, that is to say each hour will record the values, will design your table for one, two, three, and six hours respectively, that will do after breakfast, the same as for lunch and for comfort at night if you fall asleep early, take it each one hour until eight or nine at night. As soon as you get up the next day, take it on an empty stomach, so that you have an idea of ​​how your glucose transit was from the time you lay down to sleep until when you got up, because usually very little activity takes place at night. physical and it is important to take into account the necessary calories for each patient and the kind of activity performed, some patients ingest food at midnight, this of course increases their blood glucose, and if you have slow return, you will accumulate the day, sometimes breakfast at ten in the morning, lunch at twelve, take intermediate (snacks) or watch television until late at night eating high-calorie foods and therefore reaches high values.Glycemia increases as that the person with slow return of glucose ingests more food therefore, the high viscosity is what prevents the cell from nourishing, is what makes the person demand more food , feel thirsty, experience anxiety. It satisfies the stomach hunger and not the cellular hunger, consequently here the patient must do of his part, educating the appetite to avoid those excesses. There are quantities of natural products that give fluidity to the blood, in addition to others that facilitate the rapid transit of glucose in the blood. If you are unable to change your eating habits and lifestyles, you should be aware of the consequences and the ailments of this metabolic disorder. Since the problem of the slow return of glucose is not the fault of the pancreas, the therapist must focus his attention on applying procedures for liver cleansing, to allow the glucose receptors in the liver to receive excess glucose from the blood and thus not increase the blood viscosity putting the life of the patients at risk. There are many industrialized products that produce or cause this metabolic disorder, so it is important that the list also known artificial sweeteners, beverages, soft drinks, preservatives, all this affects the mechanism of the metabolic pathway of glucose in the human body bringing as a consequence its slowing, or blocking the receptors in the liver. The patient must do his part, if he is going to use the glucose metabolism clock, or if he will go to the professional therapist, but he can not forget his mental therapy in order to correct eating habits and lifestyles.

 Conclusions

 The academic world must enter to ventilate this serious error that has maintained for many decades. Although researchers have a commitment to society, it is an ethical duty, so no stipend is received because it is well understood that we have a commitment not only to the truth, but also to contribute to the welfare and development of civilization in general, In this article, in principle, it was avoided to mention the motive that has moved this whole strategy and to point out direct responsibilities, nevertheless a researcher does not ignore the geopolitics and the network ofall this emblematic issue, but it is the responsibility and duty of the universities and research centers to take the work of validating any result that has been protocolized, that is to say, one can not assign responsibility to anyone outside the academic and scientific world, if precisely the professional responsibility is to face the challenges and try to find solutions to the problems and enigmas that daily life and reality presents us with, it is the university or the academy that should be more concerned with researching since they have the resources and means available for this, so the excuses are over and they are others, they have no justification of any kind.

When an affected insulin is supplied with slow transit of glucose into the bloodstream, the glucose values ​​begin to decrease indicating that at the cellular level it accelerates glycolysis, this is deduced since with the passage of time the affected person begins to lose weight or which induces us to conclude that there is indeed little glucose from the blood to the liver, which is where the glucose receptors are (GLUT2) and therefore not transformed energy is stored in glycogen and less from the liver to muscle tissue (fat: energy reserves).

 In 1889, Josef von Mering (1849-1908) and Oskar Minkowski (1858-1931) proved that the total removal of the pancreas produced severe diabetes in the dog. This research was carried out at the Institute of Medical Clinic of the University of Strasbourg, directed by Professor B. Naunyn. We highlighted the case of Josef von Mering and Minkowski because it is presumed that it was not only a hasty diagnosis, it was also irresponsible, perhaps, to generalize it without deepen the study, and compare them with the human being, because, they ignored that the digestive process of the dog and man are totally different. To suppress a dog the pancreas where the digestion of the dog needs not only pancreatic juice, but the digestive enzymes of the pancreas since the dog swallows whole, without chewing, while the humans chew and deliver the work almost finished to the stomach. Let's think about the tremendous problem that will be presented to a dog with an amputation like these, suppressing the pancreas. These gentlemen would never ask themselves why the small intestine of the dog is relatively short compared to the human. With this we can state that the study carried out in the late nineteenth century (about thirteen decades ago) in the Medical Clinical Institute of Strasbourg was a universal disaster whose protocolization has led humanity to a historical hecatomb from the point of view of human physiology with a strong impact on anatomy as a science.

To date, 129 years later, no academic entity has demonstrated through insulinometry analysis that the little or bad use of insulin produced by the pancreas is responsible for the two types of diabetes type 1 and type 2, reiterating again that the excess glucose is removed by the liver, concluding the hypothesis that insulin is simply a positive catalyst of glycolysis since the liver is the regulator of blood glucose, that's why we are stuck in thirteen decades with the results known by all.

 By simple logic and reason, the g lucosa comes from the digestive process due to food intake, the bloodstream receives glucose as a fuel for cellular metabolism and as it uses glucose, the liver receptors are removing the excess blood, if the receptors are obstructed or there is little access, the blood glucose is dammed and its concentration increases, which gives rise to what science has called diabetes, but according to the above it is necessary to change the figure of diabetics by the expression: people with return slow glucose.

It is to note that to reach this conclusion were made analysis of cases of people diagnosed as such by conventional medicine, in the period between 2011 and 2014 in the city of Acarigua, Portuguesa state of the Republic Bolivarian of Venezuela. In the Development Development Camburito, residence of the Venezuelan citizen Cruz Elena Castro Méndez.

 It is suggested to the academic and scientific world to go to review and validate everything that has been known in this matter until today, there must be a general statement at the level globally, that is, each country through its public and private entities, medical schools and the media. In general to review what is happening inside the universities and their health programs, specializations with respect to knowledge, research processes, the teachings in the different chairs, including the chair of professional ethics and methodology of I researched itgación and very deeply in hospitals and clinics around the world in relation to their infrastructure and logistics in research and the professional performance of the physician as such.

Vocabulary:

 Catalyst [substance]: That accelerates or retards a chemical reaction without participating in it. There are two types of catalysts, the positive ones, and the negative ones: Positive catalysts: they are those that increase the speed of the reaction. Negative catalysts: also called inhibitors, are those that slow down the reaction.

 Glycolysis: Glycolysis or glycolysis is the process through which a molecule of glucose is broken down into two molecules of pyruvate. Through the glycolysis, energy is produced, which is used by the organism in different cellular processes.

 Glycolysis is generated in cells, specifically in the cytosol located in the cytoplasm. This is the most widespread procedure in all living beings, because it is generated in all types of cells, both eukaryotic and prokaryotic.

This implies that animals, plants, bacteria, fungi, algae and even protozoa organisms, are susceptible to the process of glycolysis.

 The main objective of glycolysis is to produce energy that is then used in other cellular processes of the organism.

Any diabetic solves his problem using the clock of the metabolism of glucose that was previously exposed, that is, each patient can take their treats alimnetos with differences of six hours. Have breakfast at 06:00, have lunch at 12:00 o'clock and have dinner at 6:00 o'clock, do not ingest intermediates, only water if you feel thirsty, with this we give time to the metabolism so that the transit of glucose from the blood towards the liver it is not repressed.

Bibliography of the diabetes article:

https://www.barnaclinic.com/blog/cirugia-del-pancreas/pancreas-funcion-enferma/

http://www.diabetes.org.ar/media/attachments/2018/07/11/premio-sanofi-2018.pdf

 http://hyperphysics.phy-astr.gsu.edu/hbasees/Organic/sugar.html

http://healthlibrary.uchospitals.edu/content/adult-diseases-and-conditions-v0/hand237gado-anatomand237a-y-funciones/

https://medlineplus.gov/spanish/diabetes.html

https://mx.hola.com/salud/enciclopedia-salud/2010032145258/viajes/malaria/niveles-de-glucosa-en-la-sangre/

https://www.smu.org.uy/publicaciones/libros/historicos/dm/cap1.pdf

Tejada Maury José de Jesús. Disease Metastasis of a Fraud. Cusas: Autoimmune, genetic, idiopathic or geopolitical ?. ISBN 978-958-46-2660-8. Published on August 6, 2013. Todo Artes Publicidad. Barranquilla, Colombia.

 Tejada Maury José de Jesús. Illness a Great Business, Health an Expensive Merchandise. Directed to doctors and alternative therapists. ISBN: 978-958-46-4378-0. Published April 2014. Editions Prometo S.A.S. Barranquilla, Colombia.

(*)Scientific Researcher